Conspiracy of CCSVI

the biggest scam in MS history revealed

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The Lancet: No Evidence for CCSVI in MS
Dr Zambo

Vascular aspects of multiple sclerosis

from The Lancet, Neurology, July 2011

In this Review, we have outlined three forms of vascular abnormalities that have been described in MS. Patients with MS seem to have an increased risk for ischemic stroke.
Endothelial dysfunction secondary to inflammatory responses or raised homocysteine concentrations might play a part, but reduced CBF might predispose patients to the development of these ischemic brain lesions.

The widespread cerebral hypoperfusion in MS seems not to be secondary to axonal degeneration, but might be a result of reduced axonal activity, reduced astrocyte energy metabolism, and perhaps increased blood concentrations of ET-1.

Impaired cerebral perfusion seems to be especially related to cognitive manifestations of the disease, which is a common symptom associated with substantial decline in activities of daily living. Investigation of whether interventions that improve cerebral perfusion improve cognitive function in patients with MS would be of interest.
Statements that suggest that insufficient cerebral venous drainage might play a causative part in MS have shaken both the medical and patient community. Although there is no compelling evidence to suggest that CCSVI is a cause of MS, there are some suggestions of a slower cerebral venous flow in patients with MS, which might be secondary to the reduced CBF.

There is at present no evidence for a cerebral venous outflow obstruction or stasis in MS
, and endovascular procedures for jugular vein stenoses should be undertaken only in well controlled clinical trials.


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